Are ASD symptoms evidence of a brain that has gone haywire?

Evaluating the exact triggers of symptoms that are or mimic ASD are now more than ever before under the scrutiny of “science”. So how can “science” help us to explain ASD symptoms? We can understand that there are molecular changes in the brain that are associated with ASD, including genes that have been processed, or spliced in different ways, which show changes that are implicated in a wide variety of places in the brain as compared to a neurotypical brain. We also know that scientists agree that the impact of the molecular changes in the brain not only effect regions that are relevant to language and social behavior but are found in the primary visual cortex and the parietal cortex, for example, where sensory information such as pain, temperature and touch are processed. These findings suggest an explanation for the hypersensitivity to touch and sound that is often observed in ASD patients. Scientists through patient data and computational tools, seem to conclude that there are some patterns of “dysregulation of gene expression in the frontal and temporal cortex regions of the brain.” So, we come to understand that there are complex changes, in possibly hundreds of genes, which maybe influencing one another, which can lead scientists to determine that ASD is, in the main, the result of some sort of malfunction in the brain which leads to cellular changes and dysregulation. What remains a mystery is what is causing the dysregulation to occur? If the dysregulation is due to mainly genes and infinitely small molecular changes is this a mutation from the neurotypical evolving during fetal neurodevelopment and measurable intrauterine? Does it involve complex interrelated genes or is there some other neurological system which is triggering the molecular changes which express themselves at birth or during the lifespan caused by toxins or accidents? Understanding ASD is complex and requires “science” to find the trigger.